Autopsies Of 2 Teenagers Show That Covid Jabs May Literally Be ‘Heartbreaking’

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Pathologists say that the autopsies of two teenage boys who died days after receiving their second Pfizer Covid Jabs, proves that it was the vaccine that caused their deaths.

The autopises revealed that the teenagers had ‘smoldering, catastrophic heart injuries with no symptoms’. In simple terms, the Pfizer vaccine literally broke the hearts of the two young lads who took the jabs, just like their friends did, just to get back to a normal life.

Three pathologists, two of whom are medical examiners, published their findings on Feb. 14 in an early online release article: “Autopsy Histopathologic Cardiac Findings in Two Adolescents Following the Second COVID-19 Vaccine Dose,” in the Archives of Pathology and Laboratory Medicine.

The Defender reports: The authors’ findings were conclusive. Two teenage boys were pronounced dead in their homes three and four days after receiving the second Pfizer-BioNTech COVID-19 dose.

There was no evidence of active or previous COVID-19 infection. The teens had negative toxicology screens (i.e. no drugs or poisons were present in their bodies).

These boys died from the vaccine.

Histopathological examination of their cardiac tissue revealed an important new finding: Neither heart demonstrated evidence of typical myocarditis.

Instead, the authors found evidence of microscopic changes consistent with a different form of heart injury called toxic cardiomyopathy. They wrote:

“The myocardial injury seen in these post-vaccine hearts is different from typical myocarditis and has an appearance most closely resembling a catecholamine-mediated stress (toxic) cardiomyopathy.”

The authors further explained what they observed under the microscope:

“Their histopathology does not demonstrate a typical myocarditis … In these two post-vaccination instances, there are areas of contraction bands and hypereosinophilic myocytes distinct from the inflammation.

“This injury pattern is instead similar to what is seen in the myocardium of patients who are clinically diagnosed with Takotsubo, toxic or ‘stress’ cardiomyopathy, which is a temporary myocardial injury that can develop in patients with extreme physical, chemical, or sometimes emotional stressors.

“Stress cardiomyopathy is a catecholamine-mediated ischemic process seen in high catecholamine states in the absence of coronary artery disease or spasm. It has also been called ‘neurogenic myocardial injury’ and ‘broken heart syndrome.’”

The pathologists determined there was a different mechanism of heart injury at play in these two boys, distinct from a purely infectious process that would result directly from a viral infection like COVID-19.

This is an important finding. There may be a way to distinguish cardiac injury resulting from a SARS-COV-2 infection from cardiac injury where the vaccine predisposes the patient to stress cardiomyopathy before contracting COVID-19.

However, the authors are careful not to assume that cardiac injuries from COVID-19 and COVID-19 vaccines can always be sorted out under the microscope.

They explain that stress cardiomyopathy, or “broken heart syndrome,” may also occur in a rare hyperinflammatory state that is known to occur in COVID-19 infection as well:

“This post-vaccine reaction may represent an overly exuberant immune response and the myocardial injury is mediated by similar immune mechanisms as described with SARS-COV-2 and multisystem inflammatory syndrome (MIS-C) cytokine storms.”

The authors admit this pathological finding may also occur as a result of MIS-C, a known complication of SARS-COV2 infection.

Learning more about this condition requires a biopsy of heart tissue, or in this case an autopsy. We know very little about the nature of myocarditis in people who are clinically stable because heart biopsies are not conducted on them and autopsies are rarely done on patients who die from COVID-19.

There still is no practical way of screening for cardiac injury beyond assessing symptoms.

Unfortunately, the two boys did not have symptoms of myocarditis (fever, chest pain, palpitations, or dyspnea) prior to their cardiac arrest and death. One complained of a headache and gastric upset which resolved. The other had no complaints.

This is extremely concerning. These boys had smoldering, catastrophic heart injuries with no symptoms.

How many others have insidious cardiac involvement from vaccination that won’t manifest until they get a serious case of COVID-19 or the flu? Or perhaps when they subject themselves to the physical stress of competitive sports?

These findings suggest a significant subset of COVID-19 deaths in the vaccinated could be due to the vaccines themselves.